Lengthy COVID, often known as long-haul COVID, persistent COVID or long-haul syndrome, refers to signs that persist for 4 or extra weeks after an preliminary COVID-19 an infection.1 Nonetheless, whereas this situation has primarily been seen as a aspect impact of the particular an infection, many are reporting lengthy COVID signs after getting the COVID shot as nicely,2 no matter model.
As reported by Science journal,3 “In uncommon circumstances, coronavirus vaccines might trigger lengthy COVID-like signs,” which may embody (however just isn’t restricted to) mind fog, reminiscence issues, complications, blurred imaginative and prescient, lack of odor, nerve ache, coronary heart fee fluctuations, dramatic blood stress swings and muscle weak spot. The sensation of “inner electrical shocks” are additionally reported.
The first distinction4 between post-jab lengthy COVID and lengthy COVID signs after an infection is that in individuals who get it from the an infection, early remedy was withheld and the ensuing an infection extreme. Publish-jab lengthy COVID, however, can happen both after very gentle breakthrough an infection or no breakthrough an infection in any respect.
Reluctance to Publicly Tackle Publish-Jab Lengthy COVID
In January 2021, Nationwide Institutes of Well being researchers initiated testing and tried remedy of sufferers suspected of getting lengthy COVID following their shot, however for unknown causes the investigation petered out by the top of the yr, leaving sufferers excessive and dry, with out solutions.5
Based on Science, NIH researchers did proceed their work “behind the scenes,” and different researchers, worldwide, have additionally began finding out the phenomenon. Nonetheless, there seems to be excessive reluctance to addressing post-jab lengthy COVID signs publicly. Why?
Dr. Avindra Nath, medical director on the Nationwide Institute of Neurological Problems and Stroke (NINDS) and the one main the NIH’s investigation into lengthy COVID, provides us a clue.
“Probing doable unwanted side effects presents a dilemma to researchers: They threat fomenting rejection of vaccines which are typically protected, efficient, and essential to saving lives,” Science writes.6 “‘You need to be very cautious’ earlier than tying COVID-19 vaccines to issues, Nath cautions. ‘You can also make the fallacious conclusion … The implications are enormous.’”
In different phrases, it’s all about defending the vaccine business, which has now merged with and grow to be the experimental gene remedy business.
In the meantime, the human take a look at topics are left to endure — a lot of whom don’t even understand that they ARE take a look at topics. They purchased the “protected and efficient” and “rigorously examined” lies. In Nath’s protection, he tried to publish a case collection on about 30 of those sufferers however medical journals refused to publish it.7
What’s Inflicting Lengthy COVID?
As for the mechanisms behind lengthy COVID, opinions differ. Analysis8,9 offered10 by Dr. Bruce Patterson on the Worldwide COVID Summit in Rome, in September 2021, suggests monocytes, proven to trigger lung injury in sufferers with acute COVID, are additionally concerned in lengthy COVID.
In abstract, the inflammatory cytokines which are imagined to set off T cell activation fail to take action in some folks, leading to an insufficient antiviral response. As a substitute of T cells — that are wanted to quell the an infection — B cells and a specific subset of monocytes are elevated. As described by HealthRising.org:11
“After they used antibodies to search for proof of coronavirus proteins within the monocytes … they discovered them — in spades. Seventy-three p.c of the ‘non-classical’ monocytes in long-COVID sufferers carried the coronavirus proteins …
These kind of monocytes have typically been considered anti-inflammatory, however latest research present that they’ll, in some conditions, produce pro-inflammatory cytokines. They’re largely concerned in ‘trash cleanup,’ and the antiviral response …
The authors consider these monocytes have been drawn to coronavirus-infected cells within the blood vessels, the place they ingested them, after which put a coronavirus protein on their floor to alert the immune system.
The issue in lengthy COVID happens when they’re drawn to the blood vessels and injure them, or trigger the blood vessels to inappropriately dilate.
These nonclassical monocytes are the one monocytes to hold the CX3CR1 receptor, which when it binds to fractalkine, activates an anti-apoptotic protein that permits the monocytes to outlive longer than traditional. It additionally causes the monocytes to revert from their anti-inflammatory state, and begin pumping out pro-inflammatory cytokines.
These are essential steps as most monocytes die inside a couple of days, and having very long-lived (as much as at the least 16 months) coronavirus protein-carrying monocytes is an important facet of Patterson’s speculation …
The monocyte binding additionally triggers the manufacturing of VEGF — which Patterson studies is elevated in virtually all lengthy haulers. VEGF then dilates the blood vessels inflicting, Patterson thinks, emotions of fullness within the head, migraines, and maybe cognitive issues.”
The Autoantibody Concept
One other idea, put forth by Harald Prüss, a neurologist on the German Middle for Neurodegenerative Ailments and the Charité College Hospital in Berlin, is that antibodies concentrating on the SARS-CoV-2 spike protein is likely to be inflicting “collateral injury.” As reported by Science:12
“In 2020, whereas attempting to find antibody therapies for COVID-19, [Prüss] and his colleagues found that of 18 antibodies they recognized with potent results towards SARS-CoV-2, 4 additionally focused wholesome tissues in mice — an indication they may set off autoimmune issues …
Over the previous yr, analysis teams have detected unusually excessive ranges of autoantibodies, which may assault the physique’s personal cells and tissues, in folks after a SARS-CoV-2 an infection.
In Nature in Might 2021, immunologists Aaron Ring and Akiko Iwasaki at Yale College of Medication and their colleagues reported13 discovering autoantibodies in acute COVID-19 sufferers that focus on the immune system and mind; they’re now investigating how lengthy the autoantibodies persist and whether or not they can injury tissues …
In a paper Prüss and his colleagues are about to submit, they describe discovering autoantibodies that assault mouse neurons and different mind cells in at the least one-third of these sufferers.”
Researchers are additionally investigating whether or not post-jab lengthy COVID is likely to be as a result of autoantibodies towards the angiotensin-converting enzyme 2 (ACE2) receptor,14 which is the goal of the spike protein.
Different Working Theories
Different working theories embody aberrant immune response brought on by persistent activation of a specific subset of T cells,15,16 notably in these whose lengthy COVID signs embody neurological issues.
Persistent microscopic blood clots is one other idea being labored on by Resia Pretorius, a physiologist at Stellenbosch College in South Africa.
She and her colleagues have revealed17,18 preliminary proof exhibiting microscopic blood clots can linger lengthy after the SARS-CoV-2 an infection clears. These clots then intervene with oxygen supply, which might help clarify signs reminiscent of mind fog.
One more idea is that the signs are brought on by residual spike protein lodged in your tissues and organs — together with your intestine — which may take nicely over a yr to clear after a critical an infection.19 As reported by Medical Information In the present day:20
“Researchers investigated the antigens of SARS-CoV-2 — the virus that causes COVID-19 — current in blood plasma samples collected from people with lengthy COVID and typical COVID-19 an infection.
They discovered that one explicit SARS-CoV-2 antigen — the spike protein — was current within the blood of a majority of lengthy COVID sufferers, as much as a yr after they have been first recognized with COVID-19. In sufferers with typical COVID-19 an infection, nevertheless, the spike protein was not detected.
This discovering supplies proof for the speculation that SARS-CoV-2 can persist within the physique by viral reservoirs, the place it continues to launch spike protein and set off irritation.”
In an effort to determine lengthy COVID biomarkers, the researchers measured ranges of three SARS-CoV-2 antigens: spike protein, the S1 subunit of the spike protein and the nucleocapsid (outer protein coat) of the virus.
All three antigens have been discovered within the blood of 65% of the lengthy COVID sufferers examined, however the spike protein was the most typical, and remained elevated the longest. So, briefly, an indicator of lengthy COVID is the long-term presence of spike protein, and spike protein is exactly what the COVID jabs are instructing your cells to create.
Granted, the spike protein produced by your cells in response to the shot is genetically altered, so it’s not completely similar to the spike protein discovered on SARS-CoV-2 (which by the best way additionally seems to be artifical), however no matter their supply, the spike protein seems to be a key pathogenic issue.21 As such, it is smart that many COVID jab recipients are reporting lengthy COVID-like signs, as their our bodies are frequently producing them.
mRNA Photographs Injure Hearts of ALL Recipients
Opposite to preliminary claims, we all know the mRNA within the COVID pictures journey all through the physique and accumulate in varied organs. The cells in these organs then find yourself expressing the spike protein long run.
Apart from the reproductive organs, your coronary heart is a major goal, and up to date Swiss analysis22 discovered the speed of subclinical myocarditis is a whole lot of occasions extra frequent than medical myocarditis. Apparently, whereas different research have discovered greater post-jab myocarditis charges in males, right here, it was far greater in girls.
An estimated 1 in 27 girls who obtained an mRNA COVID shot had proof of myocardial damage. What’s extra, they concluded that ALL recipients suffered some stage of coronary heart damage, even when they have been asymptomatic. Within the video above, Dr. Vinay Prasad evaluations this research and what it means to have subclinical myocarditis. As reported by The Day by day Skeptic:23
“Crucially, the research discovered elevated troponin ranges — indicating coronary heart damage — throughout all vaccinated folks … This means the vaccine is routinely injuring the guts (an organ which doesn’t heal nicely) and that the recognized accidents are simply the extra extreme cases of a far bigger quantity occurring proper throughout the board … These are usually not uncommon occasions, as is commonly claimed by medical authorities and within the media. They’re alarmingly frequent.”
COVID Jab Deaths Are Being Buried
All in all, proof reveals the COVID jabs are an absolute well being catastrophe, but our well being businesses are doing nothing to stop it. Quite the opposite, they’ve doubled and tripled down on their COVID shot suggestions whereas concurrently burying incriminating proof.
In “How FDA and CDC Are Hiding COVID Jab Risks” I element how the U.S. Meals and Drug Administration and the Facilities for Illness Management and Prevention are refusing to launch related information, have lied about trial findings, and much more egregiously, are actually manipulating databases to artificially get rid of security alerts and conceal extra jab-related deaths.
Easy methods to Deal with Lengthy COVID
Whereas remedy for post-jab accidents, which embody lengthy COVID-like signs, continues to be in its early levels, there may be hope. A variety of medical doctors, scientists and COVID specialty teams are investigating cures and dealing with affected sufferers. These embody:
• The FLCCC remedy protocol — The Frontline COVID-19 Vital Care Alliance (FLCCC) has developed protocols each for these combating lengthy COVID and people injured by the COVID jabs. You’ll be able to obtain each from covid19criticalcare.com.
• Spike protein detox — Cures that may assist inhibit, neutralize and get rid of spike protein have been recognized by the World Well being Council. Inhibitors that stop the spike protein from binding to your cells embody Prunella vulgaris, pine needle tea, emodin, neem, dandelion extract and the drug ivermectin. Dr. Pierre Kory, of FLCCC, believes ivermectin could also be the very best method to bind the circulating spike protein.
Spike protein neutralizers, which stop the spike from damaging cells, embody N-acetylcysteine (NAC), glutathione, fennel tea, star anise tea, pine needle tea, St. John’s wort, comfrey tea and vitamin C.
Time-restricted consuming (TRE) might help get rid of the poisonous proteins by stimulating autophagy, and nattokinase, a type of fermented soy, is useful for lowering blood clots, A number of further detox cures could be present in “World Council for Well being Reveals Spike Protein Detox.”
• Dietary help — “Treating Lengthy-Haul Syndrome” lists dietary dietary supplements beneficial for lengthy COVID by Dr. Al Johnson, reminiscent of vitamin C (to calm irritation), vitamin D (for total immune perform optimization), glutathione (to quell irritation) and NAC (as a precursor to glutathione).
Dr. Peter McCullough studies having had some success treating neurological signs with fluvoxamine, an SSRI antidepressant, and a March 2022 evaluate paper24 suggests combating the neurotoxic results of the spike protein utilizing the flavonoids luteolin and quercetin.
A world collaboration involving researchers in Israel and the U.S. has additionally developed what they declare is a “breakthrough” proprietary dietary formulation for lengthy COVID known as “Restore.” Examine25 outcomes recommend every of the reported signs have been alleviated in 72% to 84% of research contributors after 4 weeks of standalone use. As reported by The Jerusalem Publish:26
“The complement accommodates vitamins and plant bio-extracts for crucial immune restoration after surviving a viral an infection, with components together with zinc, vitamin D, quercetin, bromelain, St. John’s wort, Indian frankincense and beta caryophyllene, a cannabinoid CB2 agonist (agonists flip protein molecule receptors on; antagonists flip them off).”